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cover-mediumA combination of vitamin D3 and curcumin, from tumeric, may boost the immune system, and help it clear the protein plaques linked to Alzheimer’s, says a new study.

The new data, published in the Journal of Alzheimer’s Disease, may lead to new approaches in preventing Alzheimer’s by using vitamin D3 alone or in combination with natural or synthetic curcumin to boost the immune system in protecting the brain against beta-amyloid.

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Prof Martin Pall of Washington State University has been exploring the biochmical mechanisms behind the complex illness known as Multiple Chemical Sensitivity. He states: Cases of multiple chemical sensitivity (MCS) are reported to be initiated by seven classes of chemicals. Each of the seven acts along a specific pathway, indirectly producing increases in NMDA (A

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Comment: Everyone knows that vitamins “from A to zinc” are important for good health. Now, a new research study in the August 2009 print issue of the Journal of Leukocyte Biology suggests that zinc may be pointing the way to new therapeutic targets for fighting infections. Specifically, scientists from Florida found that zinc not only supports healthy immune function, but increases activation of the cells (T cells) responsible for destroying viruses and bacteria.

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plosComment: Natural Killer (NK) cells – a type of white blood cell – are a major component of the human body’s innate immune system. Over 1,000 NK cells are found in every drop of blood. They provide a fast frontline defence against tumours, viruses and bacterial infections, by latching onto and killing cells in the human body that are cancerous or are infected with a virus or a bacterial pathogen.

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coverThimerosal (ethylmercurithiosalicylic acid), an ethylmercury (EtHg)-releasing compound (49.55% mercury (Hg)), was used in a range of medical products for more than 70 years. Of particular recent concern, routine administering of Thimerosal-containing biologics/childhood vaccines have become significant sources of Hg exposure for some fetuses/infants.

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A Virus is an ultramicroscopic infectious agent.

The H1N1 A 2009 (Swine Flu)  is a novel strain of the influenza virus.

Viruses cannot reproduce on  their own. To reproduce, a virus must bind to a living cell inside some organism, insert its genetic material into that “host” cell, and take over the cell’s reproductive “machinery.” The virus then makes copies of itself – maybe hundreds of thousands. Sooner or later, this kills the infected cell – causing the virus to leave the cell and cause illness. Once out of the host cell new viruses start the process over, attacking other cells until the immune system, and or medication controls their activity and replication. The H1N1 Influenza A virus once inside a cell can produce approximately 100,000 new virions in about 8 hours.

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logo-manA mother’s exposure to urban air pollutants known as polycyclic aromatic hydrocarbons (PAHs) can adversely affect a child’s intelligence quotient or IQ, a study reports. 

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genetic changes of H1N1 virusIt is not generally appreciated that descendents of the H1N1 influenza A virus that caused the catastrophic and historic pandemic of 1918–1919 have persisted in humans for more than 90 years and have continued to contribute their genes to new viruses, causing new pandemics, epidemics, and epizootics.

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covermedInvestigators from the Centers for Disease Control and Prevention and 25 other public health agencies around the world sought to determine the genetic origin and the antigenic characteristics of the 2009 A(H1N1) influenza virus, commonly referred to as “swine flu.” Specifically, they used a genetic-similarity study to determine which influenza strains previously identified had combined to produce the new A(H1N1) flu virus.

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coverfigThe epidemic of obesity and type 2 diabetes mellitus is dramatically increasing. Environmental factors, such as sedentary life-style, hypercaloric, fat-rich diet and genetic susceptibility are considered major determinants of type 2 diabetes mellitus. Obesity and peripheral insulin resistance are hallmarks and major risk factors for development of type 2 diabetes mellitus. Cardiovascular complications (eg, atherosclerosis, coronary heart disease) of both metabolic disorders are associated with chronic subclinical inflammation.

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